Most patients who have type 2 diabetes don’t die from a high sugar. Many diabetics are not even aware their sugar is high, and they have not been diagnosed. They have no symptoms. Diabetics die of heart attacks, strokes, and kidney failure. Research has never shown that treating type 2 diabetes with insulin reduces deaths, heart attacks or stroke. Sulfonylurea drugs like glyburide lower your sugar by increasing your insulin level. They fall into the same category. In fact, in the most famous research study that looked at aggressively lowering sugar to near normal was stopped early because more patients died when compared with less aggressive care. The first clinical research study in diabetes was the University Group Diabetes Program beginning in 1961. It examined the impact of the sulfonylurea drug tolbutamide on complications of diabetes. Once again there was evidence tolbutamide caused more deaths. Like glyburide, tolbutamide works by increasing insulin levels. If diabetes is all about sugar levels, insulin and tolbutamide should reduce deaths and heart attacks, but they don’t. Diabetes specialists twist themselves into knots trying to explains this phenomenon away. It can’t be explained away because it is real.
Over the last several sessions, I have been helping you understand a larger diagram that explains how inappropriately switched-on genes make you age faster and develop chronic disease more rapidly. On the last post, I showed how you can substitute any growth factor in the EGFR position on the diagram. Insulin is well-known to be a potent growth factor. As with any other growth factor it switches on mTOR and switches off AMPK which a potent common denominator in making you age faster and have heart attacks and strokes at an earlier age.
Medications like glyburide and tolbutamide are cheap, but if they cause more people to die, they are a cure that is worse the disease. So, what’s to be done? Insulin levels go up when we eat sugar and starch—even whole grain starch. When we eat those things very consistently and in excess that persistenly raises insulin levels and activates mTOR in health. Activating mTOR creates resistance to insulin effects and insulin levels rise still further. If we do enough of that to gain weight, insulin levels and mTOR activation are persistently high. When we treat with glyburide or insulin shots, levels rise still higher. Anyone with diabetes or prediabetes should drastically restrict sugar and carbohydrate. Intermittent fasting helps. Metformin directly inhibits mTOR, activates AMPK, increases insulin sensitivity, and lowers insulin levels. If carbohydrate restriction and metformin is not enough, add a medication like Jardiance which also activates AMPK and switches off mTOR. If your sugar is still high, you are probably eating too much carbohydrate. If all that fails, high sugar over time has damaged your pancreas and the best thing is to take just enough insulin to keep your fasting glucose between 80 and 110. Diabetes is not just a sugar disease. The same biology that causes diabetes also causes artery disease and cancer. Use treatments that interfere with that core biology rather than medication that fans the flame. Your goal should be a low sugar AND a lower insulin level.
MCGDocWrites MCGDoc’s Newsletter ·just now
Finally, one of our prominent thought leaders agrees with the concept of insulin resistance! The “treatment” or “cure” is worse than the disease process, so much do they kill the treated before the disease will! The entire system of sick care is very sick itself! The Diabetic Treatments peddlers have much to answer too, to the millions of people who suffered under their “care”!!! Soul search, now! Repent! Stop lying to us!!!