A single dose of metformin cuts the size of an experimentally induced heart attack in half by switching on AMPK and increasing nitric oxide production. It does it in diabetic and non-diabetic animals. It is the signaling that matters most. What do you think.
How does the impact of metformin compare to the impact of intermittent fasting on AMPK? Is there additive value to Metformin in humans who are generally non-diabetic?
Thank you for your research, understanding and education regarding this topic!
How does the impact of metformin compare to the impact of intermittent fasting on AMPK? Is there additive value to Metformin in humans who are generally non-diabetic?